This is very, very true. I have positive confirmation.
I think you mean stigma. Unless you mean that Soy makes weeping wounds on your hands and feet.
I don’t actually find much to support this; the effect on testosterone is weaker than most people seem to think. There’s an oft-cited study (2007, Goodin et al) that found a decrease in testosterone in four weeks for a group of men fed daily soy, but it turns out that one of the men in the study had a massive decrease. Without him in the mix, it appears that Soy has an effect, but it’s nowhere near as big as some people fear. It’s easy to pick out the “odd man” in the data:
It’s possible that this was because of the soy, but it’s more likely that there were other factors at play - especially since he continued to decrease between day 28 (end of treatment) and day 42 (so that was two weeks without taking the soy.)
Also, since the study was not blinded and placebo-controlled, it’s hard to tell how much of the change in testosterone was becuase of what the men thought would happen. It’s interesting that two of the men had a solid rise in testosterone between day 14 and 28 - were they people who believed that extra protein makes them stronger, regardless? And other men who dropped during the 28 days rebounded after getting off the protein, from day 28 to day 42… but some of them rebounded to higher than their baseline. Perhaps these men were very convinced that soy would decrease their T, and were so stoked to get off the stuff, that their T rose even higher than when they started? (For reference, there’s support for the idea that hormone levels are sensitive to mood and mental status, as well as the way around, so it’s more likely to be a factor here than on average.)
As far as estrogen-mimicking effects - there are differences between a pseudo-estrogen binding to a receptor and real estrogen binding to a receptor. There’s a lot of study in the area (and it’s an important area to sort out, for diet understanding), and soy isoflavones may well turn out to have benefits and harms due to activating receptors, or due to blocking receptors that might otherwise be activated by real estrogen, or even from different mechanisms independent of estrogen-like activity.
I’m happy the science is going on actively in this area.